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The red blood of the child that is RH Positive becomes weak under the attack of antibodies. Bilirubin may collect in the brain in a condition called kernicterus. The same bilirubin (dead red blood cells) may cause jaundice. The dead red blood cells cause the liver and spleen to fill with bilirubin until the plasma of the blood seeps out of both swollen organs. The child therefore has edema and weak blood.

In a similar description of the process a RH Positive immune reaction, a child may develop Hydrops Fetalis, which is currently described as “the inability of the body to manage body fluids”. Wikipedia describes the symptoms as:

• Symptoms and signs in the fetus:

• Enlarged liver, spleen, or heart and fluid buildup in the fetus' abdomen seen via ultrasound.

• Symptoms and signs in the newborn:

• Anemia that creates the newborn's pallor (pale appearance).

• Jaundice or yellow discoloration of the newborn's skin, sclera or mucous membrane. This may be evident right after birth or after 24–48 hours after birth. This is caused by bilirubin (one of the end products of red blood cell destruction).

• Enlargement of the newborn's liver and spleen.

• The newborn may have severe edema of the entire body.

• Dyspnea (difficulty breathing).

As we study these phenomena we realize that our previously RH negative blood can become RH Positive for a variety of reasons. That means we can start making antigens we didn’t make before. If we make new antigens then we trigger a new set of antibodies and find ourselves in an autoimmune blood disease. Different organs, different systems and different problems can all arise just from activating a new set of antigens.

The term RH came from a test of a group of antigens that were tested in a Rhesus monkey. The RH disease is only for version D of 50 members of the RH Group of Antigens. The RH Antigen Blood Group represents only one group of antigens. Wikipedia identifies 35 more Antigen Blood Groups.

The picture of antigens and their responding antibodies is a much larger picture than the RH D antigen. Remember, our blood types are based on the antibodies in them. There are 108 identified blood types that have been simplified into the 8 we currently use. It makes you wonder how many antigens are in just the 35 groups of antigens that have been identified.

The RH Antigen Group deals only with blood. There is evidence that other groups of antigens attach to other parts of the body besides blood. Wherever an antigen attaches, antibodies are expected to follow.

What if antigens attached to carbohydrates? The antibodies would attack the carbohydrate and not allow the carbohydrate to be digested. We currently think that a carbohydrate-counting diet works simply because we demand less metabolism from fewer ingested carbohydrates. Wouldn’t it be interesting if the real reason is that less carbohydrates meant less swelling because of less antibodies?

What if other antibodies attached to other systems like mucous and lymph and different sugars?

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Rhesus Disease is a disease of wherein the mother’s blood antibodies attack the blood of an unborn fetus.

SUMMARY OF REMEDIES

(with range of bottles needed)

RHESUS DISEASE 6-9

The disease originally comes from a mismatch of the mother and father. Couples contemplating parenting often have the test. The RH stands for a type of antigens (target inviters for antibodies) attached to the red blood cells. If the tests show the antigens are present on the red blood cell, the person is RH positive. If the red blood cells do not have the RH antigens, the person is RH negative. If both prospective parents are RH negative the test is good. If one or both of the prospective tests are positive, there is a strong change of blood problems with the child.

Even when the couple tests compatible, if the mother is exposed to the fetus’ blood during pregnancy, her system can turn to RH Positive. In other words, if the mother’s blood encounters the fetus blood, the mother’s blood may make the RH Positive antigens, which makes the child RH Positive. RH Positive means that the antibodies that answer the RH Positive targets will cause inflammatory reactions for the mother and the child. If a mix of the blood comes during delivery, the mother may make RH positive antigens and the second and subsequent children may be at risk. Therefore, the second or subsequent children are considered more likely to be afflicted than the first child. The disease is more common among Caucasians than any other race. 13% of Caucasian women give birth to a first child and become RH Positive from the mixed blood, even though parents started as RH Negative.

The disease is considered rare, but our research challenges the rarity. In severe cases currently acknowledged by tests, the child dies unless given multiple, complete blood transfusions. Less severe cases produce brain damage and multiple blood weaknesses. Organ function, hormone production, immunity, digestion, mental clarity and personality disorders are showing probable links to “subclinical” cases of the RH Disease. The discovery of the mix of blood causing RH Positive (antigen) factors may explain why this may be more common than thought.

We see that the blood can become RH Positive from operations, blood infections and even blood diseases. We also see that an RH Positive factor can be transferred by saliva and/or sexual fluids.

One child in Canada tested to be severely deficient in almost everything. He fit no pattern we knew until we used this remedy. The mother, a medical doctor, says the child is improving steadily. One of our practitioners took the remedy for a short period as an experiment. Her menstrual problems cleared significantly in less than a bottle.

17% of the pregnant mothers in Australia have this disease diagnosed. In a heart warming story, they found one man who has a natural antibody to the disease. Since he was age 18, every week James Harrison has donated blood to make vaccines for the disease. He is considered a national hero in Australia for saving the lives of 2,200,000 babies. James is 78 in 2015 and by law can only donate blood up to age 80. Since he started donating, 50 other people in Australia have been found with the rare antibody.

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