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Respiratory alkalosisEffects of resp alkalosisSelf perpetuating process: hyperV removes CO2 cerebral acidosis further incr in RRChronic resp alkalosis is unique in that it can have full metabolic compensation (within 4/7)Met: HYPOcalcaemia, HYPOkalaemia, HYPOphosphatemia tetany and carpopedal spasm; incr metabolic demand of tissuesNS: decr seizure thresholdCV: vasoC, decr myocardial O2 delivery, incr myocardial O2 demandRS: incr pul cap perm, decr pul compliance, worsens intrapul shunt, incr airway resistance; Shift 02 dissociation curve to the left (Alkalosis) (Increased affinity of Hb for O2)CausesStimulated respiratory driveCNS CVA, ICH, psychogenic, cerebral oedema, hepatic encephalopathyHypermetabolic Thyrotoxicosis, Pregnancy (Progesterone)(Secondary to reduced FRC), Sepsis (fever) (often before metabolic acidosis), DT, anxiety, pain, DKA and aspirin ODEnvironmental HYPERthermia (Heat tetany), altitude related, exerciseDrugs Aspirin OD, ammonia, progesterone, theophylline, CO, stimulantsIatrogenic Mechanical ventilationHypoxemia inducedPneumonia, PE, asthma, Congenital heart disease, Chronic altitude compensation, Early in altitude acclimatisation, pul oedemaCompensation for metabolic acidosisCompensationAcute HCO3 decr 2 per 10 change in CO2 from 40 2:10 Decr HCO3 expected = ( (40 – pCO2) X 2 ) 210Chronic Only process that can have full compensation HCO3 decr 5 per 10 change in CO2 from 40 5:10 Decr HCO3 expected = ( (40 – pCO2) X 5 ) 210Notes from: Dunn, Cameron ................
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