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More recently, it has been proposed that liver fibrosis and hepatic cirrhosis depend on the balance between the classical (ACE-Ang Ⅱ-AT. 1. receptor) and the counter-regulatory (ACE2-Ang-(1-7)-Mas receptor) RAS axes[16,28,64-67]. In cirrhotic patients and rats with liver fibrosis, the balance between both RAS axes also accounts for ... ................
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